Metatron 4025 Hunter - Prevention And Treatment Of Pancreatitis
Pancreatitis is a disease of the pancreas caused by the self-digesting effect of trypsin. The pancreas has edema, congestion, or bleeding or necrosis. Clinically, symptoms such as abdominal pain, bloating, nausea, vomiting, and fever appear. Elevated levels of amylase in blood and urine, etc. It can be divided into acute and chronic. We can use Metatron 4025 Hunter to instantly healthy pancreas status
Most of them are sudden onset, manifested as severe upper abdominal pain, which radiates to the shoulders and backs, and the patients consciously feel a "banded feeling" in the upper abdomen and lower back. In the case of edematous pancreatitis, the abdominal pain is mostly persistent with paroxysmal aggravation. Acupuncture or injection of antispasmodic drugs can relieve the abdominal pain; in the case of hemorrhagic pancreatitis, the abdominal pain is very severe and often accompanied by shock. It is difficult to relieve pain with general pain relief methods. Nausea and vomiting appear at the beginning of the onset, and its characteristic is that the abdominal pain cannot be relieved after vomiting. The frequency of vomiting is also consistent with the severity of the lesion. In edematous pancreatitis, there is not only nausea, but also often vomiting for 1 to 3 times; in hemorrhagic pancreatitis, vomiting is severe or persistent and frequent retching. May have fever, jaundice, etc. The degree of fever is mostly consistent with the severity of the lesion. Edema pancreatitis may have no fever or only mild fever; hemorrhagic necrotizing pancreatitis may have high fever. If the fever does not go away, complications may occur, such as pancreatic abscess. The occurrence of jaundice may be caused by concurrent biliary tract disease or by compression of the common bile duct by the enlarged head of the pancreas.
A very small number of patients have a very sudden onset, and may have no obvious symptoms or soon after the onset of symptoms, that is, shock or death occurs, which is called sudden death or fulminant pancreatitis.
It varies in severity. There may be no obvious clinical symptoms, and there may be obvious various clinical manifestations. Up to 90% of patients have abdominal pain of varying degrees, which occurs once every few months or years, and is persistent pain. Severe pain is accompanied by nausea and vomiting. Abdominal pain in these patients often has postural characteristics. The patient likes to curl up, sitting or leaning forward, and the abdominal pain worsens when lying down or standing upright. Mild patients have no symptoms of diarrhea, but severely ill patients have excessive acinar destruction and decreased secretion, and symptoms appear. Manifestations of abdominal distension and diarrhea, stool 3 to 4 times a day, large volume, light color, shiny and air bubbles on the surface, foul smell, mostly acid reaction, the patient appears weight loss, weakness and malnutrition and other manifestations.
Some dyspeptic symptoms such as bloating, decreased appetite, nausea, fatigue, weight loss and other symptoms are common in patients with severely impaired pancreatic function. Such as pancreatic islet involvement can significantly affect glucose metabolism, and about 10% have obvious symptoms of diabetes. In addition, patients with biliary diseases or biliary obstruction may have jaundice. Abdominal masses can be palpable in those with pseudocyst formation. A small number of patients may have pancreatic ascites. In addition, chronic pancreatitis can cause upper gastrointestinal bleeding. Patients with chronic pancreatitis can develop multiple fat necrosis. Subcutaneous fat necrosis often occurs in the extremities and can form hard nodules under the skin.
Acute pancreatitis diagnosis
Mainly based on clinical manifestations, related laboratory examinations and imaging examinations, clinically require not only the diagnosis of pancreatitis, but also the evaluation of its disease development, complications and prognosis. All patients with upper abdominal pain should think of the possibility of acute pancreatitis. This one is the prerequisite for the diagnosis of acute pancreatitis. Especially when the diagnosis of upper abdominal pain is not clear or the administration of antispasmodic analgesics cannot be relieved, it is more likely to be pancreatitis.
The diagnosis of this disease should meet the following 4 criteria:
(1) Have typical clinical manifestations, such as upper abdominal pain or nausea and vomiting, accompanied by upper abdominal tenderness or peritoneal irritation;
(2) There is an increase in pancreatin content in serum, urine or abdominal puncture fluid;
(3) Image inspection (ultrasound, CT) shows pancreatitis or pancreatitis as seen by surgery or autopsy pathology;
(4) Other diseases with similar clinical manifestations can be excluded.
Chronic pancreatitis diagnosis
The clinical manifestations are variable and non-specific, and the diagnosis is often difficult. Atypical patients are more difficult to make a clear diagnosis. For patients with recurrent acute pancreatitis, biliary disease or diabetes, recurrent or persistent upper abdominal pain, chronic diarrhea, weight loss cannot be explained by other diseases, and this disease should be suspected. Clinical diagnosis is mainly based on medical history, physical examination and supplemented by necessary X-ray, ultrasound, NLS diagnosis(Metatron 4025 Hunter) or other imaging examinations, upper gastrointestinal endoscopy and related laboratory examinations. The latest diagnostic criteria for chronic pancreatitis (Japanese Society of Pancreatology, 1995) are as follows:
Criteria for diagnosis of chronic pancreatitis
(1) There are pancreatic stones in the abdominal B-ultrasound pancreatic tissue.
(2) CT intrapancreatic calcification was confirmed with pancreatic stones.
(3) ERCP: The pancreatic duct and its branches in the pancreatic tissue are irregularly expanded and unevenly distributed; the main pancreatic duct is partially or completely blocked and contains pancreatic stones or protein emboli.
(4) Secretion test: The secretion of bicarbonate is reduced, and the secretion or excretion of pancreatin is reduced.
(5) Histological examination: The tissue section shows the destruction and reduction of pancreatic exocrine tissue, and there is irregular fibrosis between the lobules, but the fibrosis between the lobules is not unique to chronic pancreatitis.
(6) Duct epithelial hyperplasia or dysplasia, cyst formation.
Acute edematous pancreatitis is mainly treated with palliative treatment, while hemorrhagic necrotizing pancreatitis should be treated according to the situation. In the course of non-surgical treatment of acute edematous pancreatitis, it is necessary to closely observe the evolution of its course. The treatment of localized pancreatic necrosis is still controversial.
Most of the preoperative preparations for acute edematous pancreatitis and hemorrhagic necrotic pancreatitis, non-surgical treatments include: prevention and treatment of shock, improvement of microcirculation, antispasmodic, pain relief, inhibition of pancreatic enzyme secretion, anti-infection, nutritional support, and prevention of complications Happened and so on.
Surgical methods include: pancreatic capsule incision and decompression; pancreatic necrosis tissue debridement; regular pancreatectomy; open abdomen obstruction; abdomen installation zipper operation and so on.
1. Abdominal pain treatment:
General treatment: Patients with chronic pancreatitis must absolutely abstain from alcohol and avoid overeating. Use caution with certain drugs that may be related to the disease: sulfasalazine, estrogen, glucocorticoids, indomethacin, hydrochlorothiazide, methyldopa, etc.
The main methods are:
Endoscopic stent placement and pancreatic duct sphincterotomy
2. Treatment of steatorrhea: Limit fat intake. The degree of limitation depends on the severity of fat malabsorption, generally less than 20g per day. If fat intake is not effective, medical treatment such as medicine must be started.
3. Diabetes treatment: Diabetes often occurs in patients with severe advanced chronic pancreatitis, and it may appear only when more than 80% of the pancreatic tissue is destroyed. Control your diet and cooperate with pancreatin to enhance the absorption of fat and protein.
4. Meta Therapy Through Metatron 4025 Hunter: Meta Therapy has good effects in the prevention and adjuvant treatment of pancreatitis.
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