Metatron Hunter 4025 And Atrophic Gastritis

Atrophic gastritis, also known as chronic atrophic gastritis, is characterized by atrophy of the epithelium and glands of the gastric mucosa, reduced number, thinning of the gastric mucosa, thickening of the base layer of the mucosa, or accompanied by pyloric and intestinal metaplasia, or atypical hyperplasia Characteristic chronic digestive system diseases. It often manifests as dull pain, fullness, belching, loss of appetite, weight loss, anemia, etc. in the upper abdomen, without specificity. It is a disease with multiple pathogenic factors and precancerous lesions. Through Metatron Hunter 4025, the stomach can be detected better and faster, to detect gastric diseases in advance and treat gastritis in advance.

Causes of atrophic gastritis

1. Helicobacter pylori (Hp) infection

Hp can be cultured in the gastric mucosa of 60% to 90% of patients with chronic gastritis. In 1986, the eighth meeting of the World Gastroenterology Society proposed that Hp infection is one of the important causes of chronic gastritis.

2. Eating habits

Smoking, drinking, food irritation, drugs that damage the gastric mucosa, etc.

3. Immune factors

In atrophic gastritis, especially in the blood, gastric juice or plasma cells of atrophic mucosa, antibodies to parietal cells or intrinsic factor antibodies can often be found in patients with gastric gastritis. Therefore, autoimmune reactions are considered to be the cause of atrophic gastritis.

4. Bile or duodenal fluid reflux.

5. Physical factors

Clinical statistics show that the occurrence of this disease is significantly positively correlated with age. The older the age, the worse the gastric mucosal function "resistance", and it is susceptible to damage caused by adverse external factors.

6. Genetic factors

The position in the incidence of type A atrophic gastritis has been confirmed. The positive rate of PCA and IFA is high in family members of pernicious anemia, and atrophic gastritis is common.

7. Metal contact

Lead workers have a high incidence of gastric ulcer, and gastric mucosal biopsy revealed an increase in the incidence of atrophic gastritis. In addition to lead, many heavy metals such as mercury, copper and zinc have certain damaging effects on the gastric mucosa.

8.Radiation

Radiation therapy for ulcer disease or other tumors can damage or even shrink the gastric mucosa.

9. Iron deficiency anemia

Many facts show that iron deficiency anemia is closely related to atrophic gastritis.

10. Other

The continuation of chronic superficial gastritis and so on.

The clinical manifestations of atrophic gastritis are not only lack of specificity, but also not completely consistent with the extent of the disease. The following clinical manifestations often occur: gastric cavity fullness; gastric cavity pain; heartburn and indigestion symptoms; stool abnormalities and weakness; anemia.

The symptoms and signs of atrophic gastritis are non-specific and cannot be used as a basis for diagnosis. The diagnosis is mainly based on gastroscopy and gastric mucosal biopsy.

(1) Most of them are middle-aged or older, with a long course of disease, and often have a history of chronic superficial gastritis.

(2) Symptoms and signs: Long-term indigestion, fullness and discomfort of the gastric cavity, anorexia, fatigue, weight loss, anemia, etc.

(3) Gastroscopy

①The color of gastric mucosa becomes lighter;

②Submucosal blood vessels can be seen through;

③The mucosal folds are small or even disappear;

④When atrophic gastritis is accompanied by hyperplasia of glands and neck or intestinal epithelial metaplasia, the surface of the mucosa is rough and uneven, granular or nodular, and sometimes pseudopolyps can be seen, and the features of submucosal blood vessels are often hidden;

⑤ The fragility of the atrophic mucosa increases, it is easy to bleed, and there may be erosions;

⑥Atrophic gastritis may be accompanied by chronic superficial gastritis, such as hyperemia, erythema, adhesion of mucus, and increased reflection.

(4) Pathological examination

① Atrophy of the inherent glands in the gastric mucosa;

② Metaplasia;

③ Hyperplasia;

④ Cancer.

(5) Laboratory inspection

① Gastric juice analysis Type A CAG patients are mostly acid-free or low-acid, while type B CAG patients can be normal or low-acid;

② Determination of pepsinogen. Pepsinogen is secreted by the principal cells. In atrophic gastritis, the content of pepsinogen in blood and urine decreases;

③ Serum gastrin was measured to secrete gastrin by G cells in the mucosa of gastric antrum. In patients with type A CAG, serum gastrin is often significantly increased; in patients with type B CAG, gastric antral mucosa atrophy, which directly affects the function of gastrin secretion by G cells, and serum gastrin is lower than normal;

④Immunological examination of parietal cell antibodies, intrinsic factor antibodies, and gastrin secreting cell antibodies can be used as an auxiliary diagnosis for atrophic gastritis and its classification.

(6) NLS diagnosis of Metatron Hunter 4025

Research the stomach organs, then perform Entropy analysis and NLS analysis.

Atrophic gastritis treatment

The symptoms and signs of atrophic gastritis are non-specific and cannot be used as a basis for diagnosis. The diagnosis is mainly based on gastroscopy and gastric mucosal biopsy.

(1) Most of them are middle-aged or older, with a long course of disease, and often have a history of chronic superficial gastritis.

(2) Symptoms and signs: Long-term indigestion, fullness and discomfort of the gastric cavity, anorexia, fatigue, weight loss, anemia, etc.

(3) Gastroscopy

①The color of gastric mucosa becomes lighter;

②Submucosal blood vessels can be seen through;

③The mucosal folds are small or even disappear;

④When atrophic gastritis is accompanied by hyperplasia of glands and neck or intestinal epithelial metaplasia, the surface of the mucosa is rough and uneven, granular or nodular, and sometimes pseudopolyps can be seen, and the features of submucosal blood vessels are often hidden;

⑤ The fragility of the atrophic mucosa increases, it is easy to bleed, and there may be erosions;

⑥Atrophic gastritis may be accompanied by chronic superficial gastritis, such as hyperemia, erythema, adhesion of mucus, and increased reflection.

(4) Pathological examination

① Atrophy of the inherent glands in the gastric mucosa;

② Metaplasia;

③ Hyperplasia;

④ Cancer.

(5) Laboratory inspection

① Gastric juice analysis Type A CAG patients are mostly acid-free or low-acid, while type B CAG patients can be normal or low-acid;

② Determination of pepsinogen. Pepsinogen is secreted by the principal cells. In atrophic gastritis, the content of pepsinogen in blood and urine decreases;

③ Serum gastrin was measured to secrete gastrin by G cells in the mucosa of gastric antrum. In patients with type A CAG, serum gastrin is often significantly increased; in patients with type B CAG, gastric antral mucosa atrophy, which directly affects the function of gastrin secretion by G cells, and serum gastrin is lower than normal;

④Immunological examination of parietal cell antibodies, intrinsic factor antibodies, and gastrin secreting cell antibodies can be used as an auxiliary diagnosis for atrophic gastritis and its classification.

The clinical manifestations of atrophic gastritis not only lack specificity, but are not completely consistent with the extent of the disease, so early detection and treatment are necessary, Metatron Hunter 4025 can help you do this. At the same time Meta Therapy can help you for treatment and improvement.